Gracias al grupo de Facebook de OMA, una de las magníficas mamás que está ahí siempre dando su apoyo, Jane, he obtenido la informacción de este sitio, la Asociación Americana de Oftalmología Pediatrica y Estrabismo, que está en San Francisco (lo cierto es que en USA los científicos y médicos están haciendo una muy buena labor y al menos encuentro información acerca de la apraxia ocular..en cambio aquí....uffff).
La he copiado tal cual y os dejo el link, es muy fácil de traducir, y es lo que hay. Aunque debo decir que nada de lo que dicen es desconocido pues ya había encontrado informes de diferentes procedencias y todos coinciden. Los que leéis este blog lo sabéis. De todos modos aquí está muy bien explicada, pero por supuesto se debe esperar a que haya más investigaciones y debe leerse con precaución, entendiendo que no se trata en ningún momento de un diagnóstico para quien padezca apraxia ocular..
Espero que dejen de tratar a mi hijo como un vago, el es lento porque la apraxia ocular es así, pero hay que darle tiempo.
La información puede asustar de entrada, pero lo cierto es que los niños con apraxia pueden avanzar y mejorar mucho, la prueba está en facebook, y creo que la causa de las mejoras es el amor y la dedicación.
Oculomotor Apraxia
What is oculomotor apraxia?
Saccades are quick, simultaneous movements of both eyes in the same direction and are abnormal in the condition of oculomotor apraxia (OMA). Most children with OMA must utilize a head thrust to initiate horizontal eye movement away from the straight-ahead gaze position. Typically, vertical eye movements are unaffected by the condition.
Where is the problem with this condition, in the eyes or in the brain?
The source of OMA is in the brain. The process of initiating eye movements is a complicated neural pathway involving many different structures. Neuroimaging with magnetic resonance imaging (MRI) is commonly performed when evaluating OMA. Findings may be normal or may reveal poor development of regions of the brain, in particular the corpus callosum, cerebellum, and/or fourth ventricle.
What other developmental problems can coexist with OMA?
Extensive investigations sometimes reveal no associated developmental issues. However, children with OMA often have developmental delays and possess low muscle tone (hypotonia). Speech, reading, and motor delays are common even when neuroimaging studies are normal.
What other clinical associations can exist with OMA?
The wide range of clinical entities that have been reported in children with OMA include: agenesis of the corpus callosum, Joubert syndrome, Dandy-Walker malformation, microcephaly, hydrocephalus, vermis hypoplasia, porencephalic cyst, megalocephaly, Krabbe’s leucodystrophy, Pelizaeus Merzbacher disease, Infantile Gaucher disease, GM1 gangliosidosis, Infantile Refsum’s disease, propionic academia, ataxia telangiectasia, Bardet-Biedl syndrome, vermis astrocytoma, vermis cyst, carotid fibromuscular hypoplasia, Cornelia de Lange syndrome, and microphthalmos.
What causes OMA to develop?
The etiology of OMA is usually not determined and considered idiopathic. However, the condition is sometimes considered secondary and attributed to insults occurring either during the perinatal period or the first 6 months of life. Associations may include perinatal hypoxia, meningitis, periventricular leucomalacia, cerebral palsy, septicemia, anemia, herpes encephalitis, and seizure disorder.
If a child has OMA, do siblings or future children have a risk for the condition?
There are no reports of siblings with OMA. It would be extremely rare if other children were born with the same condition.
What is the treatment for OMA?
There is no specific treatment for OMA. Serial ophthalmologic examinations are recommended to monitor for other eye problems associated with OMA.
Does OMA improve over time and resolve?
There are few reports of the long-term prognosis of children born with OMA. The head thrusts associated with OMA typically diminish over time, but tend not to completely disappear. This may represent a true improvement in the disorder or be an adaptive compensatory mechanism to mask the head thrust.
